Correction: Ferroportin Gene Polymorphism, Ferritin, and TIBC Levels Associated with the Severity of COVID-19 among patients: A Sequencing Analysis

Abstract

This Correction refers to: Ban Adnan Hatem, Ferdous A Jabir, (2025). Ferroportin Gene Polymorphism, Ferritin, and TIBC Levels Associated with the Severity of COVID-19 among patients: A Sequencing Analysis. Advancements in Life Sciences, 12(4). https://doi.org/10.62940/als.v12i4.2150

Following an internal post-publication quality review, the Editorial Office identified citation mismatches in the above article. The authors have reviewed the highlighted references and confirmed the following corrections. These changes concern the accuracy and appropriateness of the cited references only and do not affect the study design, experimental dataset, statistical analysis, results, tables, figures, or principal conclusions of the study.

Reference corrections

• Ref. [3] Replaced with new Ref. [3] (Ganz, 2013), below. The statement concerning ferroportin-mediated iron efflux from macrophages is now supported by an appropriate reference addressing systemic iron homeostasis, ferroportin-mediated iron export, and macrophage iron recycling.
• Ref. [5] Replaced with new Ref. [5] (Ganz, 2011), below. The statement concerning hepcidin-mediated iron trapping in enterocytes, macrophages, and hepatocytes is now supported by a reference specifically addressing the hepcidinferroportin pathway and iron sequestration.
• Ref. [9] Replaced with new Ref. [9] (Hickman et al., 2000), below. The statement regarding unsaturated iron-binding capacity (UIBC) as a screening approach for iron overload/hemochromatosis is now supported by a study evaluating UIBC as a screening strategy for hereditary hemochromatosis.
• Ref. [10] Removed. The corresponding sentence describes the objective of the present study rather than a previously established scientific finding. The sentence has been revised as follows:

Therefore, the purpose of this study was to investigate the relationship between COVID-19 severity and Ferroportin, ferritin, and TIBC levels, and to explore the presence of Ferroportin gene polymorphisms.

Corrected references

Ref. [3]. Ganz T. Systemic iron homeostasis. Physiological Reviews, (2013); 93(4): 17211741. doi:10.1152/physrev.00008.2013.

Ref. [5]. Ganz T. Hepcidin and iron regulation, 10 years later. Blood, (2011); 117(17): 44254433. doi:10.1182/blood-2011-01-258467.

Ref. [9]. Hickman PE, Hourigan LF, Powell LW, Cordingley F, Dimeski G, Ormiston B, Shaw J, Ferguson W, Johnson M, Ascough J, McDonell K, Pink A, Crawford DHG. Automated measurement of unsaturated iron binding capacity is an effective screening strategy for C282Y homozygous haemochromatosis. Gut, (2000); 46(3): 405409. doi:10.1136/gut.46.3.405.

These corrections do not affect the study's data, results, statistical outcomes, or conclusions.

Correction Published

03 October 2026: The necessary corrections have been carried out in the Version of Record.